癌细胞组成的植物

Wogonin导致肿瘤细胞的死亡程序凋亡，而对健康细胞几乎没有影响。 Scientists from the German Cancer Research Center (Deutsches Krebsforschungszentrum, DKFZ) have found the molecular mechanism underlying this selectivity.来自德国癌症研究中心（Deutsches Krebsforschungszentrum，DKFZ）的科学家发现了这种选择性的分子机制。 The mechanism underlying the selective effect of this plant constituent had still been unclear.尚不清楚该植物成分的选择性作用的机制。 There are two different ways by which the apoptosis program can be started in a cell: by external stimuli or by signals from within the cell as a response to factors such as radioactive radiation or reactive oxygen compounds such as hydrogen peroxide (H2O2).可以通过两种不同的方式启动细胞凋亡程序：通过外部刺激或通过细胞内信号响应诸如放射性辐射或活性氧化合物（如过氧化氢（HXNUMXOXNUMX））的反应。 Defects in genes that control growth can turn a cell into a threat for the whole organism.控制生长的基因缺陷可以将细胞变成对整个生物体的威胁。 Defective cells that might get out of control are driven into suicide by a protective mechanism called apoptosis.可能失控的缺陷细胞被称为凋亡的保护机制驱使自杀。 However, this life saving mechanism is no longer working in most tumor cells, since numerous molecules regulating apoptosis are defective.然而，由于许多调节细胞凋亡的分子是有缺陷的，因此这种挽救生命的机制在大多数肿瘤细胞中不再起作用。